Hyalinised fibrocartilaginous tumour of bone: probable new histological entity with FOSL1 gene rearrangement

Fernanda Amary; Roberto Tirabosco; Jane Chalker; Suzanne MacMahon; Adrienne Flanagan; Paul O'Donnell

doi:10.1111/his.15462

journal article Apr 27, 2025

Hyalinised fibrocartilaginous tumour of bone: probable new histological entity with FOSL1 gene rearrangement

Fernanda Amary

Roberto Tirabosco

Jane Chalker Suzanne MacMahon Adrienne Flanagan

Paul O'Donnell

Histopathology Vol. 87 No. 1 pp. 157-159 · Wiley

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References

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10.1097/pas.0000000000000469

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Details

Published: Apr 27, 2025
Vol/Issue: 87(1)
Pages: 157-159
License: View

Authors

F

Fernanda Amary

Cellular and Molecular Pathology Royal National Orthopaedic Hospital NHS Trust Middlesex UK; Research Department of Pathology University College London Cancer Institute London UK

R

Roberto Tirabosco

Cellular and Molecular Pathology Royal National Orthopaedic Hospital NHS Trust Middlesex UK; Research Department of Pathology University College London Cancer Institute London UK

J

Jane Chalker

Clinical Genomics Department The Centre for Molecular Pathology, The Royal Marsden NHS Foundation Trust Sutton UK

S

Suzanne MacMahon

Clinical Genomics Department The Centre for Molecular Pathology, The Royal Marsden NHS Foundation Trust Sutton UK

A

Adrienne Flanagan

Cellular and Molecular Pathology Royal National Orthopaedic Hospital NHS Trust Middlesex UK; Research Department of Pathology University College London Cancer Institute London UK

P

Paul O'Donnell

Research Department of Pathology University College London Cancer Institute London UK; Clinical Radiology Royal National Orthopaedic Hospital NHS Trust Middlesex UK

Cite This Article

Fernanda Amary, Roberto Tirabosco, Jane Chalker, et al. (2025). Hyalinised fibrocartilaginous tumour of bone: probable new histological entity with FOSL1 gene rearrangement. Histopathology, 87(1), 157-159. https://doi.org/10.1111/his.15462

Hyalinised fibrocartilaginous tumour of bone: probable new histological entity with FOSL1 gene rearrangement

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